The Protein Kinase Cδ Catalytic Fragment Targets Mcl-1 for Degradation to Trigger Apoptosis

This publication has 52 references indexed in Scilit:

• N-Benzyladriamycin-14-Valerate (AD198) Induces Apoptosis through Protein Kinase C-δ–Induced Phosphorylation of Phospholipid Scramblase 3
  Cancer Research, 2005
• Bax Activation and Induction of Apoptosis in Human Keratinocytes by the Protein Kinase C δ Catalytic Domain
  Journal of Investigative Dermatology, 2004
• Protein Kinase Cδ Regulates Keratinocyte Death and Survival by Regulating Activity and Subcellular Localization of a p38δ-Extracellular Signal-Regulated Kinase 1/2 Complex
  Molecular and Cellular Biology, 2004
• Low‐dose UV‐radiation sensitizes keratinocytes to TRAIL‐induced apoptosis
  Journal of Cellular Physiology, 2004
• Activation of Caspase-9 Is Required for UV-induced Apoptosis of Human Keratinocytes
  Journal of Biological Chemistry, 2002
• Differences in DNA binding properties between E2F1 and E2F4 specify repression of the Mcl-1 promoter
  Oncogene, 2002
• Direct repression of the Mcl-1 promoter by E2F1
  Oncogene, 2002
• Caspase activation and disruption of mitochondrial membrane potential during UV radiation-induced apoptosis of human keratinocytes requires activation of protein kinase C
  Cell Death & Differentiation, 2002
• The Proteolytic Cleavage of Protein Kinase C Isotypes, Which Generates Kinase and Regulatory Fragments, Correlates with Fas‐Mediated and 12‐O‐Tetradecanoyl‐Phorbol‐13‐Acetate‐Induced Apoptosis
  European Journal of Biochemistry, 1997
• Normal keratinization in a spontaneously immortalized aneuploid human keratinocyte cell line.
  The Journal of cell biology, 1988