Interplay between RORγt, Egr3, and E Proteins Controls Proliferation in Response to Pre-TCR Signals
Open Access
- 1 June 2006
- Vol. 24 (6) , 813-826
- https://doi.org/10.1016/j.immuni.2006.03.023
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health
- American Cancer Society
This publication has 48 references indexed in Scilit:
- Over‐expression of Aurora‐A targets cytoplasmic polyadenylation element binding protein and promotes mRNA polyadenylation of Cdk1 and cyclin B1Genes to Cells, 2005
- The BCL2A1 gene as a pre–T cell receptor–induced regulator of thymocyte survivalThe Journal of Experimental Medicine, 2005
- An essential function for the nuclear receptor RORγt in the generation of fetal lymphoid tissue inducer cellsNature Immunology, 2003
- T cell stimulation in the absence of exogenous antigen: a T cell signal is induced by both MHC-dependent and -independent mechanismsEuropean Journal of Immunology, 2003
- E-proteins directly regulate expression of activation-induced deaminase in mature B cellsNature Immunology, 2003
- Retinoid-related orphan receptors (RORs): roles in cell survival, differentiation and diseaseCell Death & Differentiation, 2002
- Regulation of the TCRα repertoire by the survival window of CD4+CD8+ thymocytesNature Immunology, 2002
- Identification of unknown target genes of human transcription factors using chromatin immunoprecipitationMethods, 2002
- Induction of the Early Growth Response (Egr) Family of Transcription Factors during Thymic SelectionThe Journal of Experimental Medicine, 1997
- Control points in early T-cell developmentImmunology Today, 1993