Novel Anion Dependence of Induced Cation Transport in Malaria-infected Erythrocytes
Open Access
- 1 October 1995
- journal article
- research article
- Published by Elsevier
- Vol. 270 (41) , 24270-24275
- https://doi.org/10.1074/jbc.270.41.24270
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Nutrient transport pathways in Plasmodium-infected erythrocytes: What and where Are They?Parasitology Today, 1994
- Glibenclamide and meglitinide block the transport of low molecular weight solutes into malaria‐infected erythrocytesFEBS Letters, 1993
- Altered membrane permeability: a new approach to malaria chemotherapyParasitology Today, 1992
- The increased K+ leak of malaria-infected erythrocytes is not via a Ca2+-activated K+ channelBiochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1992
- Nitrendipine is a potent inhibitor of the Ca2+‐activated K+ channel of human erythrocytesFEBS Letters, 1992
- Transport of lactate inPlasmodium falciparum-infected human erythrocytesJournal of Cellular Physiology, 1991
- Chloride channels of biological membranesBiochimica et Biophysica Acta (BBA) - Reviews on Biomembranes, 1990
- Intercellular adhesion molecule-1 is an endothelial cell adhesion receptor for Plasmodium falciparumNature, 1989
- Biophysical analysis of novel transport pathways induced in red blood cell membranesThe Journal of Membrane Biology, 1987
- New permeability pathways induced in membranes of Plasmodium falciparum infected erythrocytesMolecular and Biochemical Parasitology, 1983