Abstract
Three articles in this issue of the Journal discuss new information about the acquired immunodeficiency syndrome (AIDS). One reviews the immune abnormalities in the disease and their relation to immunodeficiencies in other human disorders and in animal models.1 The other two focus on the association of a human retrovirus, human T-cell lymphotropic virus/lymphadenopathy-associated virus (HTLV-III/LAV), with the disease.2 , 3 Like the discovery of the causes of legionnaires' disease and Lyme arthritis, the rapid association of HTLV-III/LAV infection4 , 5 with AIDS soon after the disease was first described is a triumph of today's biologic science. It is also evidence that society now has . . .