Dissociation of oxidant production by peroxisome proliferator-activated receptor ligands from cell death in human cell lines
- 1 July 2004
- journal article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 37 (1) , 36-47
- https://doi.org/10.1016/j.freeradbiomed.2004.04.015
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Troglitazone but not rosiglitazone induces G1 cell cycle arrest and apoptosis in human and rat hepatoma cell linesToxicology Letters, 2003
- PPARα and the regulation of cell division and apoptosisToxicology, 2002
- Cytotoxic Effect of Peroxisome Proliferator Fenofibrate on Human HepG2 Hepatoma Cell Line and Relevant MechanismsToxicology and Applied Pharmacology, 2002
- Activation of Peroxisome Proliferator-Activated Receptor-g Inhibits Apoptosis Induced by Serum Deprivation in LLC-PK1 CellsNephron Experimental Nephrology, 2002
- Cellular response to oxidative stress: Signaling for suicide and survival*Journal of Cellular Physiology, 2002
- PPAR? agonists inhibit cell growth and suppress the expression of cyclin D1 and EGF-like growth factors in ras-transformed rat intestinal epithelial cellsInternational Journal of Cancer, 2001
- Inhibition of peroxisome-proliferator-activated receptor (PPAR)α by MK886Biochemical Journal, 2001
- Troglitazone Suppresses Cell Growth of Myeloid Leukemia Cell Lines by Induction of p21WAF1/CIP1 Cyclin-Dependent Kinase InhibitorBiochemical and Biophysical Research Communications, 1999
- Control of the peroxisomal β-oxidation pathway by a novel family of nuclear hormone receptorsPublished by Elsevier ,1992
- Activation of a member of the steroid hormone receptor superfamily by peroxisome proliferatorsNature, 1990