A Comparative Study of Apoptosis and Necrosis in HepG2 Cells: Oxidant-Induced Caspase Inactivation Leads to Necrosis

This publication has 34 references indexed in Scilit:

• Apoptosis: A Modulator of Cellular Homeostasis and Disease States
  Annals of the New York Academy of Sciences, 1998
• Calpain Activation and Not Oxidative Damage Mediatesl-2-Chloropropionic Acid-Induced Cerebellar Granule Cell Necrosis
  Toxicology and Applied Pharmacology, 1997
• Involvement of Cellular Proteolytic Machinery in Apoptosis
  Biochemical and Biophysical Research Communications, 1997
• Resolution of acute inflammation and the role of apoptosis in the tissue fate of granulocytes
  Clinical Science, 1992
• Plasma Membrane Bleb Formation and Rupture: A Common Feature of Hepatocellular Injury
  Hepatology, 1990
• Cell Death Induced in a Murine Mastocytoma by 42–47°C Heatingin Vitro: Evidence that the Form of Death Changes from Apoptosis to Necrosis Above a Critical Heat Load
  International Journal of Radiation Biology, 1990
• The formation of plasma membrane blebs in hepatocytes exposed to agents that increase cytosolic Ca²⁺ is mediated by the activation of a non‐lysosomal proteolytic system
  FEBS Letters, 1986
• Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation
  Nature, 1980
• Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics
  British Journal of Cancer, 1972
• Shrinkage necrosis: A distinct mode of cellular death
  The Journal of Pathology, 1971