The platelet cyclooxygenase metabolite 12-L-hydroxy-5, 8, 10- hepta- decatrienoic acid (HHT) may modulate primary hemostasis by stimulating prostacyclin production
- 1 November 1987
- journal article
- research article
- Published by Elsevier in Prostaglandins
- Vol. 34 (5) , 749-763
- https://doi.org/10.1016/0090-6980(87)90297-8
Abstract
No abstract availableThis publication has 29 references indexed in Scilit:
- 12-L-hydroxy-5,8,10-heptadecatrienoic acid (HHT) is an excellent substrate for NAD+-dependent 15-hydroxyprostaglandin dehydrogenaseBiochemical and Biophysical Research Communications, 1985
- Increased Prostacyclin Biosynthesis in Patients with Severe Atherosclerosis and Platelet ActivationNew England Journal of Medicine, 1984
- Prostacyclin and thromboxane in diabetes.BMJ, 1981
- In-vitro venous prostacyclin production, plasma 6-keto-prostaglandin F1 alpha concentrations, and diabetic retinopathy.BMJ, 1981
- Release of Prostaglandins and Thromboxane into the Coronary Circulation in Patients with Ischemic Heart DiseaseNew England Journal of Medicine, 1981
- Prostacyclin production by cultured smooth muscle cells from atherosclerotic rabbit aortaNature, 1980
- Stimulation of phosphatidic acid production in platelets precedes the formation of arachidonate and parallels the release of serotoninBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1979
- Conversion of prostaglandin endoperoxides to C17‐hydroxy acids catalyzed by human platelet thromboxane synthaseFEBS Letters, 1977
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976
- A RAPID METHOD OF TOTAL LIPID EXTRACTION AND PURIFICATIONCanadian Journal of Biochemistry and Physiology, 1959