Prenatal nicotine exposure impairs β-adrenergic function: Persistent chronotropic subsensitivity despite recovery from deficits in receptor binding
- 1 August 1990
- journal article
- research article
- Published by Elsevier in Brain Research Bulletin
- Vol. 25 (2) , 233-237
- https://doi.org/10.1016/0361-9230(90)90066-9
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Prenatal exposure to nicotine impairs nervous system development at a dose which does not affect viability or growthBrain Research Bulletin, 1989
- Impaired ontogeny of striatal dopamine D1 and D2 binding sites after postnatal treatment of rats with SCH-23390 and spiroperidolDevelopmental Brain Research, 1989
- Effects of prenatal nicotine exposure on neuronal development: Selective actions on central and peripheral catecholaminergic pathwaysBrain Research Bulletin, 1987
- Nicotine administration to rats: Methodological considerationsLife Sciences, 1987
- Effects of maternal nicotine injections on brain development in the rat: Ornithine decarboxylase activity, nucleic acids and proteins in discrete brain regionsBrain Research Bulletin, 1986
- Development of α‐Noradrenergic and Dopaminergic Receptor Systems Depends on Maturation of Their Presynaptic Nerve Terminals in the Rat BrainJournal of Neurochemistry, 1981
- EFFECTS OF MATERNAL CIGARETTE SMOKING ON THE FETUS AND PLACENTABJOG: An International Journal of Obstetrics and Gynaecology, 1978
- Smoking in Pregnancy and Subsequent Child DevelopmentBMJ, 1973
- Cigarette Smoking and Plasma Levels of NicotineNature, 1972
- A method of stimulating different segments of the autonomic outflow from the spinal column to various organs in the pithed cat and ratBritish Journal of Pharmacology, 1970