MUTAGENICITY OF THE OPTICAL ISOMERS OF THE DIASTEREOMERIC BAY-REGION CHRYSENE 1,2-DIOL-3,4-EPOXIDES IN BACTERIAL AND MAMMALIAN-CELLS

  • 1 January 1982
    • journal article
    • research article
    • Vol. 42  (8) , 2972-2976
Abstract
The mutagenic activities of the 4 optically pure (+)- and (.sbd.)-enantiomers of the 2 diastereomeric bay-region chrysene 1,2-diol-3,4-epoxides [carcinogen] were evaluated in histidine-dependent strains of Salmonella typhimurium and in cultured Chinese hamster V79 cells. In strain TA98 of S. typhimurium, (.sbd.)-1.alpha.,2.beta.-dihydroxy-3.beta.,4.beta.-epoxy-1,2,3,4-tetrahydrochrysene was 5-10 times more active than the other 3 optical isomers. In strain TA100 of S. typhimurium and in Chinese hamster V79 cells, (+)-1.beta.,2.alpha.-dihydroxy-3.alpha.,4.alpha.-epoxy-1,2,3,4-tetrahydrochrysene was the most mutagenic diol-epoxide and was from 5-40 times more active than the other 3 optical isomers. The bay-region (+)- and (.sbd.)-3,4-epoxy-1,2,3,4-tetrahydrochrysene isomers had identical mutagenic activities in all 3 systems. The presence and orientation of the hydroxyl groups may play an important role in modulating the mutagenic activity of bay-region epoxides of chrysene in both bacterial and mammalian cells.

This publication has 19 references indexed in Scilit: