Deactivation of Vasodilator Responses by GRK2 Overexpression:A Mechanism or the Mechanism for Hypertension?
Open Access
- 1 April 2002
- journal article
- Published by Elsevier in Molecular Pharmacology
- Vol. 61 (4) , 707-709
- https://doi.org/10.1124/mol.61.4.707
Abstract
No abstract availableThis publication has 15 references indexed in Scilit:
- Vascular-Targeted Overexpression of G Protein-Coupled Receptor Kinase-2 in Transgenic Mice Attenuates β-Adrenergic Receptor Signaling and Increases Resting Blood PressureMolecular Pharmacology, 2002
- β-arrestin- and c-Src-dependent degradation of G-protein-coupled receptor kinase 2The EMBO Journal, 2001
- The β 2 -Adrenergic Receptor Delivers an Antiapoptotic Signal to Cardiac Myocytes Through G i -Dependent Coupling to Phosphatidylinositol 3′-KinaseCirculation Research, 2000
- G-Protein–Coupled Receptor Kinase Activity in HypertensionHypertension, 2000
- G Protein-coupled Receptor Kinase 5 in Cultured Vascular Smooth Muscle Cells and Rat AortaPublished by Elsevier ,1997
- G-protein-coupled receptor kinase activity is increased in hypertension.Journal of Clinical Investigation, 1997
- Enhanced Expression of Gi-Protein Precedes the Development of Blood Pressure in Spontaneously Hypertensive RatsJournal of Molecular and Cellular Cardiology, 1997
- G-Protein Function Is Reduced in HypertensionHypertension, 1997
- G Protein Alterations in Hypertension and AgingHypertension, 1995
- Effect of Dichloroisoproterenol on Vascular Responses to Catecholamines in Man*Journal of Clinical Investigation, 1964