Mutator phenotype of mammalian cells due to deficiency of NEIL1 DNA glycosylase, an oxidized base-specific repair enzyme
- 20 May 2008
- journal article
- Published by Elsevier in DNA Repair
- Vol. 7 (8) , 1213-1220
- https://doi.org/10.1016/j.dnarep.2008.03.025
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Identification and characterization of a human DNA glycosylase for repair of modified bases in oxidatively damaged DNAProceedings of the National Academy of Sciences, 2002
- hMYH cell cycle-dependent expression, subcellular localization and association with replication foci: evidence suggesting replication-coupled repair of adenine:8-oxoguanine mispairsNucleic Acids Research, 2001
- Abortive base-excision repair of radiation-induced clustered DNA lesions in Escherichia coliProceedings of the National Academy of Sciences, 2001
- A reliable assessment of 8-oxo-2-deoxyguanosine levels in nuclear and mitochondrial DNA using the sodium iodide method to isolate DNANucleic Acids Research, 2001
- Evolution of instability at coding and non-coding repeat sequences in human MSI-H colorectal cancers.Human Molecular Genetics, 2001
- Germline and Somatic Mutation Analysis of MLH3 in MSI-Positive Colorectal CancerThe American Journal of Pathology, 2000
- Base excision repair of DNA in mammalian cellsFEBS Letters, 2000
- Mmh / Ogg1 gene inactivation results in accumulation of 8-hydroxyguanine in miceProceedings of the National Academy of Sciences, 2000
- The DNA repair gene MBD4 (MED1) is mutated in human carcinomas with microsatellite instabilityNature Genetics, 1999
- Conditional Mutator Phenotypes in hMSH2-Deficient Tumor Cell LinesScience, 1997