Thyrotropin-releasing hormone activates a Ca2+-dependent polyphosphoinositide phosphodiesterase in permeable GH3 cells. GTP gamma S potentiation by a cholera and pertussis toxin-insensitive mechanism.

This publication has 61 references indexed in Scilit:

• Direct stimulation by thyrotropin-releasing hormone (TRH) of polyphosphoinositide hydrolysis in GH3 cell membranes by a guanine nucleotide-modulated mechanism
  Biochemical and Biophysical Research Communications, 1985
• Prolactin secretion in permeable GH3 pituitary cells is stimulated by Ca2+ and protein kinase C activators
  Biochemical and Biophysical Research Communications, 1985
• Potential role for a guanine nucleotide regulatory protein in chemoattractant receptor mediated polyphosphoinositide metabolism, Ca++ mobilization and cellular responses by leukocytes
  Biochemical and Biophysical Research Communications, 1985
• Stepwise enzymatic dephosphorylation of inositol 1,4,5-trisphosphate to inositol in liver
  Nature, 1984
• Polyphosphoinositide hydrolysis by phospholipase C is accelerated by thyrotropin releasing hormone (TRH) in clonal rat pituitary cells (GH₃ cells)
  FEBS Letters, 1984
• Chemotactic factor causes rapid decreases in phosphatidylinositol,4,5-bisphosphate and phosphatidylinositol 4-monophosphate in rabbit neutrophils
  Biochemical and Biophysical Research Communications, 1983
• Calcium-dependence of catecholamine release from bovine adrenal medullary cells after exposure to intense electric fields
  The Journal of Membrane Biology, 1982
• Phosphatidylinositol specific phospholipases C
  Life Sciences, 1982
• Guanine nucleotides modulate trh-receptor binding in sheep anterior pituitary
  Molecular and Cellular Endocrinology, 1981
• Inositol phospholipids and cell surface receptor function
  Biochimica et Biophysica Acta (BBA) - Reviews on Biomembranes, 1975