Abstract
The multiorgan failure that is the hallmark of severe sepsis results from the activation of a very wide range of mediator cascades, including cytokines, complement, coagulation factors and others. Recently, nitric oxide has emerged as yet another component of this highly complex system. Knowledge of the basic pathophysiological processes involved in sepsis continues to grow, although the clinical applications remain uncertain.
Keywords

This publication has 0 references indexed in Scilit: