We need to inhibit complement in glomerular proteinuria
- 1 December 1999
- journal article
- Published by Elsevier in Kidney International
- Vol. 56 (6) , 2314-2315
- https://doi.org/10.1046/j.1523-1755.1999.00804.x
Abstract
No abstract availableKeywords
This publication has 9 references indexed in Scilit:
- Crry, a complement regulatory protein, modulates renal interstitial disease induced by proteinuriaKidney International, 1999
- Transgenic Mice Overexpressing the Complement Inhibitor Crry as a Soluble Protein Are Protected from Antibody-induced Glomerular InjuryThe Journal of Experimental Medicine, 1998
- Antisense oligodesoxynucleotide strategies in renal and cardiovascular diseaseKidney International, 1998
- The role of complement in the pathogenesis of tubulointerstitial lesions in rat mesangial proliferative glomerulonephritis.Journal of the American Society of Nephrology, 1997
- Amelioration of lupus-like autoimmune disease in NZB/WF1 mice after treatment with a blocking monoclonal antibody specific for complement component C5.Proceedings of the National Academy of Sciences, 1996
- Experimental insights into the tubulointerstitial disease accompanying primary glomerular lesions.Journal of the American Society of Nephrology, 1994
- Localization of the complement regulatory proteins in the normal human kidneyKidney International, 1994
- Alternative pathway activation of complement by cultured human proximal tubular epithelial cellsKidney International, 1994
- Tubulointerstitial Changes as a Major Determinant in the Progression of Renal DamageAmerican Journal of Kidney Diseases, 1992