C-reactive protein: a surrogate risk marker or mediator of atherothrombosis?

Abstract
Accumulating laboratory data suggest that CRP may directly elicit a variety of proatherogenic effects in vascular tissue. However, further research is required before we can conclude that these effects are clinically relevant. As will be discussed below, the concentrations of CRP required to elicit some of these proatherogenic effects may be greater than those used for clinical cardiovascular risk prediction.

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