The GluR5 subtype of kainate receptor regulates excitatory synaptic transmission in areas CA1 and CA3 of the rat hippocampus
- 1 October 1998
- journal article
- research article
- Published by Elsevier in Neuropharmacology
- Vol. 37 (10-11) , 1269-1277
- https://doi.org/10.1016/s0028-3908(98)00148-8
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Regulation of glutamate release by presynaptic kainate receptors in the hippocampusNature, 1996
- Selective antagonism of AMPA receptors unmasks kainate receptor-mediated responses in hippocampal neuronsNeuron, 1995
- Kainate-binding proteins: phytogeny, structures and possible functionsTrends in Pharmacological Sciences, 1994
- Cyclothiazide reverses AMPA receptor antagonism of the 2,3-benzodiazepine, GYKI 53655European Journal of Pharmacology: Molecular Pharmacology, 1993
- GYKI 52466, a 2,3-benzodiazepine, is a highly selective, noncompetitive antagonist of AMPA/kainate receptor responsesNeuron, 1993
- Cloning of a novel glutamate receptor subunit, GluR5: Expression in the nervous system during developmentNeuron, 1990
- Ibotenic Acid Analogues. Synthesis, Molecular Flexibility, and in Vitro Activity of Agonists and Antagonists at Central Glutamic Acid ReceptorsJournal of Medicinal Chemistry, 1985
- On the relationship between kainic acid-induced epileptiform activity and hippocampal neuronal damageNeuropharmacology, 1981
- Synaptic localization of kainic acid binding sitesNature, 1980
- Intraventricular kainic acid preferentially destroys hippocampal pyramidal cellsNature, 1978