Nanoparticles-A Thoracic Toxicology Perspective

This publication has 110 references indexed in Scilit:

• Single-Walled Carbon Nanotube Induces Oxidative Stress and Activates Nuclear Transcription Factor-κB in Human Keratinocytes
  Nano Letters, 2005
• Ultrafine carbon black particles stimulate proliferation of human airway epithelium via EGF receptor-mediated signaling pathway
  American Journal of Physiology-Lung Cellular and Molecular Physiology, 2004
• Ultrafine Particles Exert Prothrombotic but Not Inflammatory Effects on the Hepatic Microcirculation in Healthy Mice In Vivo
  Circulation, 2004
• Effects of Diesel Exhaust Particles on the Release of Interleukin-1 and Tumor Necrosis Factor-Alpha from Rat Alveolar Macrophages
  Experimental Lung Research, 1997
• Pulmonary Inflammatory, Chemokine, and Mutagenic Responses in Rats after Subchronic Inhalation of Carbon Black
  Toxicology and Applied Pharmacology, 1996
• Biopersistence of Man-Made Vitreous Fibers and Crocidolite Asbestos in the Rat Lung Following Inhalation
  Fundamental and Applied Toxicology, 1996
• Particle Overload in Toxicological Studies: Friend or Foe?
  Journal of Aerosol Medicine, 1990
• Paradoxical Vasoconstriction Induced by Acetylcholine in Atherosclerotic Coronary Arteries
  New England Journal of Medicine, 1986
• Prevention of coronary thrombosis with subthrombolytic doses of tissue-type plasminogen activator.
  Circulation, 1985
• The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine
  Nature, 1980