A Novel Role for FAK as a Protease-Targeting Adaptor Protein
- 1 August 2003
- journal article
- Published by Elsevier in Current Biology
- Vol. 13 (16) , 1442-1450
- https://doi.org/10.1016/s0960-9822(03)00544-x
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- Cutting to the chase: calpain proteases in cell motilityTrends in Cell Biology, 2002
- Cleavage of Focal Adhesion Kinase by Different Proteases during Src-regulated Transformation and ApoptosisPublished by Elsevier ,2001
- Epidermal Growth Factor Receptor Activation of Calpain Is Required for Fibroblast Motility and Occurs via an ERK/MAP Kinase Signaling PathwayJournal of Biological Chemistry, 2000
- Degraded Collagen Fragments Promote Rapid Disassembly of Smooth Muscle Focal Adhesions That Correlates with Cleavage of Pp125FAK, Paxillin, and TalinThe Journal of cell biology, 1999
- Src family kinases are required for integrin but not PDGFR signal transductionThe EMBO Journal, 1999
- EGF receptor regulation of cell motility: EGF induces disassembly of focal adhesions independently of the motility-associated PLCγ signaling pathwayJournal of Cell Science, 1998
- Calpain Regulation of Cytoskeletal Signaling Complexes in Von Willebrand Factor-stimulated PlateletsPublished by Elsevier ,1997
- Focal adhesion kinase (pp125FAK) cleavage and regulation by calpainBiochemical Journal, 1996
- Reduced cell motility and enhanced focal adhesion contact formation in cells from FAK-deficient miceNature, 1995
- Colocalization of calcium-dependent protease II and one of its substrates at sites of cell adhesionCell, 1987