Elevated sympathetic activity may promote insulin resistance syndrome by activating alpha-1 adrenergic receptors on adipocytes
- 1 May 2004
- journal article
- Published by Elsevier in Medical Hypotheses
- Vol. 62 (5) , 830-838
- https://doi.org/10.1016/j.mehy.2003.11.007
Abstract
No abstract availableKeywords
This publication has 128 references indexed in Scilit:
- Hypothesis: β-Adrenergic Receptor Blockers and Weight GainHypertension, 2001
- Protein Kinase C Modulates the Insulin-Stimulated Increase in Akt1 and Akt3 Activity in 3T3-L1 AdipocytesBiochemical and Biophysical Research Communications, 1998
- The N-terminal Region of the Third Intracellular Loop of the Parathyroid Hormone (PTH)/PTH-related Peptide Receptor Is Critical for Coupling to cAMP and Inositol Phosphate/Ca2+ Signal Transduction PathwaysJournal of Biological Chemistry, 1996
- The effect of dietary salt on insulin sensitivityEuropean Journal of Clinical Investigation, 1995
- Exercise training lowers resting renal but not cardiac sympathetic activity in humans.Hypertension, 1991
- Threonine 1336 of the human insulin receptor is a major target for phosphorylation by protein kinase CBiochemistry, 1990
- Pharmacological evidence for involvement of the sympathetic nervous system in the increase in renin secretion produced by a low sodium diet in ratsLife Sciences, 1990
- Sympathetic stimulation and hypertension in the pyridoxine-deficient adult rat.Hypertension, 1988
- Sympathetic outflow in human muscle nerves increases during hypoglycemiaDiabetes, 1986
- Transient enhancement of sympathetic nervous system activity by long-term restriction of sodium intake.Circulation, 1985