Apoptosis in substantia nigra following developmental hypoxic-ischemic injury
Open Access
- 1 December 1995
- journal article
- Published by Elsevier in Neuroscience
- Vol. 69 (3) , 893-901
- https://doi.org/10.1016/0306-4522(95)00282-n
Abstract
No abstract availableKeywords
Funding Information
- Parkinson's Disease Foundation
This publication has 32 references indexed in Scilit:
- Evidence of Nuclear DNA Fragmentation Following Hypoxia‐Ischemia in the Infant Rat Brain, and Transient Forebrain Ischemia in the Adult GerbilBrain Pathology, 1994
- Neonatal hypoxic-ischemic or excitotoxic striatal injury results in a decreased adult number of substantia nigra neuronsNeuroscience, 1992
- Ultrastructural changes in the hippocampal CA1 region following transient cerebral ischemia: evidence against programmed cell deathExperimental Brain Research, 1992
- Internucleosomal DNA Cleavage Should not be the Sole Criterion for Identifying ApoptosisInternational Journal of Radiation Biology, 1992
- Preserved striatal tyrosine hydroxylase activity, assessed in vivo, following neonatal hypoxia-ischemiaDevelopmental Brain Research, 1991
- Unilateral hypoxic-ischemic injury in neonatal rat results in a persistent increase in the density of striatal tyrosine hydroxylase immunoperoxidase stainingDevelopmental Brain Research, 1991
- Trophic factors and neuronal survivalNeuron, 1989
- The functional anatomy of basal ganglia disordersTrends in Neurosciences, 1989
- Glutamate neurotoxicity and diseases of the nervous systemNeuron, 1988
- A Reliable and Sensitive Method to Localize Terminal Degeneration and Lysosomes in the Central Nervous SystemStain Technology, 1980