Systemic administration of lithium chloride and tacrine increases nitric oxide synthase activity in the hippocampus of rats
- 11 June 1993
- journal article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 237 (1) , 61-64
- https://doi.org/10.1016/0014-2999(93)90093-w
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- The neuroprotective effect of a nitric oxide inhibitor in a rat model of focal cerebral ischaemiaBritish Journal of Pharmacology, 1992
- Tacrine-induced seizures and brain damage in LiCl-treated rats can be prevented by Nω-nitro-L-arginine methyl esterEuropean Journal of Pharmacology, 1992
- Tacrine: A pharmacological reviewProgress in Neurobiology, 1991
- Nitric oxide synthase protein and mRNA are discretely localized in neuronal populations of the mammalian CNS together with NADPH diaphoraseNeuron, 1991
- Glutamate, nitric oxide and cell-cell signalling in the nervous systemTrends in Neurosciences, 1991
- Localization of nitric oxide synthase indicating a neural role for nitric oxideNature, 1990
- Receptor-mediated generation of an EDRF-like intermediate in a neuronal cell line detected by spin trapping techniquesBiochemical and Biophysical Research Communications, 1990
- Chronic lithium treatment and status epilepticus induced by lithium and pilocarpine cause selective changes of amino acid concentrations in rat brain regionsNeurochemical Research, 1989
- The pharmacology of cholinergic excitatory responses in hippocampal pyramidal cellsBrain Research, 1984
- Systemic Cholinergic Agents Induce Seizures and Brain Damage in Lithium-Treated RatsScience, 1983