Estrogen Receptor, Cyclic Adenosine Monophosphate, and Protein Kinase A Are Involved in the Nongenomic Pathway by Which Estradiol Accelerates Oviductal Oocyte Transport in Cyclic Rats1
Open Access
- 1 April 2003
- journal article
- Published by Oxford University Press (OUP) in Biology of Reproduction
- Vol. 68 (4) , 1225-1231
- https://doi.org/10.1095/biolreprod.102.011395
Abstract
This investigation examined the role of estrogen receptor (ER) on the stimulatory effect of estradiol (E2) on protein phosphorylation in the oviductKeywords
This publication has 45 references indexed in Scilit:
- Invited Review: Cell localization, physiology, and nongenomic actions of estrogen receptorsJournal of Applied Physiology, 2001
- Membrane-associated binding sites for estrogen contribute to growth regulation of human breast cancer cellsOncogene, 2001
- Rapid actions of plasma membrane estrogen receptorsTrends in Endocrinology & Metabolism, 2001
- Mechanisms of Estrogen ActionPhysiological Reviews, 2001
- Rapid, Nongenomic Steroid Actions: A New Age?Frontiers in Neuroendocrinology, 2000
- Rapid effects of estrogen to modulate G protein-coupled receptors via activation of protein kinase A and protein kinase C pathwaysSteroids, 1999
- Estrogen action via the cAMP signaling pathway: stimulation of adenylate cyclase and cAMP-regulated gene transcription.Proceedings of the National Academy of Sciences, 1994
- Progesterone Abbreviates the Nuclear Retention of Estrogen Receptor in the Rat Oviduct and Counteracts Estrogen Action on Egg Transport1Biology of Reproduction, 1988
- Ovum Transport in Pregnant Rats is Little Affected by RU486 and Exogenous Progesterone as Compared to Cycling Rats1Biology of Reproduction, 1987
- Ovum Transport and Fertility following Postovulatory Treatment with Estradiol in RatsBiology of Reproduction, 1979