Direct current cardioversion does not cause cardiac damage: evidence from cardiac troponin T estimation
Open Access
- 1 September 1998
- Vol. 80 (3) , 229-230
- https://doi.org/10.1136/hrt.80.3.229
Abstract
Aim To determine whether elective direct current (dc) cardioversion of atrial fibrillation/flutter causes myocardial damage. Methods and results Cardiac troponin T and creatine kinase were estimated 20–28 hours after dc cardioversion in 51 patients who received dc shocks for elective cardioversion of chronic atrial fibrillation/flutter. Although creatine kinase was raised in 44 patients, cardiac troponin T was undetectable in all patients. Conclusion Cardiac damage does not occur as a result of cardioversion.Keywords
This publication has 16 references indexed in Scilit:
- To T or Not to T, That Is the QuestionClinical Chemistry, 1997
- Cardiac Troponin I Does Not Increase After CardioversionChest, 1997
- Measurement of Serum Troponin T, Creatine Kinase MB Isoenzyme, and Total Creatine Kinase following Arduous Physical TrainingAnnals of Clinical Biochemistry: International Journal of Laboratory Medicine, 1995
- Cardioversion of atrial fibrillationPublished by Oxford University Press (OUP) ,1995
- Troponin T for the Differential Diagnosis of Ischaemic Myocardial DamageAnnals of Clinical Biochemistry: International Journal of Laboratory Medicine, 1993
- Serum cardiac troponin T after extraordinary endurance exerciseThe Lancet, 1992
- Cardiac Troponin T in the Diagnosis of Myocardial InjuryCritical Reviews in Clinical Laboratory Sciences, 1992
- Incidence and clinical significance of ST segment elevation after electrical cardioversion of atrial fibrillation and atrial flutterAmerican Heart Journal, 1991
- Metabolic Changes and Mitochondrial Dysfunction Early Following Transthoracic Countershock in DogsPacing and Clinical Electrophysiology, 1989
- Death and damage caused by multiple direct current shocks: studies in an animal modelEuropean Heart Journal, 1988