Acute inhibition of myostatin-family proteins preserves skeletal muscle in mouse models of cancer cachexia
- 1 January 2010
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 391 (3) , 1548-1554
- https://doi.org/10.1016/j.bbrc.2009.12.123
Abstract
No abstract availableKeywords
Funding Information
- American Cancer Society Research (TBE-111831)
- ACS Institutional
- Papanicoulau Corps for Cancer Research
- Women’s Cancer Association of the University of Miami, the Wendy Will Case Cancer Fund
- Department of Defense Breast Cancer Research Program
- The Leadership Alliance Summer Research Early Identification Program
This publication has 36 references indexed in Scilit:
- Pretreatment with a soluble activin type IIB receptor/Fc fusion protein improves hypoxia-induced muscle dysfunctionAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2010
- The effects of a soluble activin type IIB receptor on obesity and insulin sensitivityInternational Journal of Obesity, 2009
- Inhibition of myostatin does not ameliorate disease features of severe spinal muscular atrophy miceHuman Molecular Genetics, 2009
- Muscle myostatin signalling is enhanced in experimental cancer cachexiaEuropean Journal of Clinical Investigation, 2008
- Analyzing real-time PCR data by the comparative CT methodNature Protocols, 2008
- Genetic Analysis of the Role of Proteolysis in the Activation of Latent MyostatinPLOS ONE, 2008
- Sprinting without myostatin: a genetic determinant of athletic prowessTrends in Genetics, 2007
- Interleukin-6 inhibits oxidative injury and necrosis after extreme liver resectionHepatology, 2007
- Lack of myostatin results in excessive muscle growth but impaired force generationProceedings of the National Academy of Sciences, 2007
- Muscular atrophy of caveolin-3–deficient mice is rescued by myostatin inhibitionJournal of Clinical Investigation, 2006