Increased lipolytic activity and high ratio of free fatty acids to albumin in sera from women with preeclampsia leads to triglyceride accumulation in cultured endothelial cells

This publication has 8 references indexed in Scilit:

• Sera from preeclamptic women increase the content of triglycerides and reduce the release of prostacyclin in cultured endothelial cells
  Thrombosis Research, 1991
• Impaired vasodilation of forearm resistance vessels in hypercholesterolemic humans.
  Journal of Clinical Investigation, 1990
• Lipolyzed hypertriglyceridemic serum and triglyceride-rich lipoprotein cause lipid accumulation in and are cytotoxic to cultured human endothelial cells. High density lipoproteins inhibit this cytotoxicity
  Thrombosis Research, 1990
• Preeclampsia: An endothelial cell disorder
  American Journal of Obstetrics and Gynecology, 1989
• At physiologic albumin/oleate concentrations oleate uptake by isolated hepatocytes, cardiac myocytes, and adipocytes is a saturable function of the unbound oleate concentration. Uptake kinetics are consistent with the conventional theory.
  Journal of Clinical Investigation, 1989
• Distinct murine macrophage receptor pathway for human triglyceride-rich lipoproteins.
  Journal of Clinical Investigation, 1988
• Enhanced macrophage degradation of biologically modified low density lipoprotein.
  Arteriosclerosis: An Official Journal of the American Heart Association, Inc., 1983
• The hyperlipidemia of pregnancy in normal and complicated pregnancies
  American Journal of Obstetrics and Gynecology, 1979