c-Src Kinase Activity Is Required for Hepatocyte Growth Factor-induced Motility and Anchorage-independent Growth of Mammary Carcinoma Cells
Open Access
- 1 December 1998
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 273 (50) , 33714-33721
- https://doi.org/10.1074/jbc.273.50.33714
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Phosphatidylinositol 3-Kinase Activity Is Required for Hepatocyte Growth Factor-induced Mitogenic Signals in Epithelial CellsJournal of Biological Chemistry, 1996
- Specific Uncoupling of GRB2 from the Met ReceptorJournal of Biological Chemistry, 1996
- Pathways Downstream of Shc and Grb2 Are Required for Cell Transformation by the Tpr-Met OncoproteinJournal of Biological Chemistry, 1996
- Hepatocyte growth factor/scatter factor induces a variety of tissue-specific morphogenic programs in epithelial cells.The Journal of cell biology, 1995
- Hepatocyte Growth Factor-induced Scatter of Madin-Darby Canine Kidney Cells Requires Phosphatidylinositol 3-KinaseJournal of Biological Chemistry, 1995
- c-Src enhances the spreading of src-/- fibroblasts on fibronectin by a kinase-independent mechanism.Genes & Development, 1995
- Overexpression of theMET/HGF receptor in ovarian cancerInternational Journal of Cancer, 1994
- Capacity of adipose tissue to promote growth and metastasis of a murine mammary carcinoma: Effect of estrogen and progesteroneInternational Journal of Cancer, 1992
- Hepatocyte growth factor has potent anti‐proliferative activity in various tumor cell linesFEBS Letters, 1991
- Association between the PDGF receptor and members of the src family of tyrosine kinasesCell, 1990