N-acetyltransferase 2 (NAT2) genotypes, cigarette smoking, and the risk of breast cancer
- 31 July 2004
- journal article
- research article
- Published by Elsevier in Cancer Detection Prevention
- Vol. 28 (3) , 187-193
- https://doi.org/10.1016/j.cdp.2004.04.001
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- NAT2 slow acetylation and GSTM1 null genotypes may increase postmenopausal breast cancer risk in long-term smoking womenPharmacogenetics, 2003
- Association of NAT2 and smoking in relation to breast cancer incidence in a population-based case-control study (United States).Cancer Causes & Control, 2003
- Breast cancer and active and passive smoking: the role of the N-acetyltransferase 2 genotype.American Journal of Epidemiology, 2000
- Association betweenN-acetyltransferase 2 (NAT2) genetic polymorphism and development of breast cancer in post-menopausal Chinese women in Taiwan, an area of great increase in breast cancer incidenceInternational Journal of Cancer, 1999
- DNA adducts of heterocyclic amine food mutagens: implications for mutagenesis and carcinogenesisCarcinogenesis: Integrative Cancer Research, 1999
- Association Between Glutathione S-Transferase M1, P1, and T1 Genetic Polymorphisms and Development of Breast CancerJNCI Journal of the National Cancer Institute, 1998
- A prospective study of NAT2 acetylation genotype, cigarette smoking, and risk of breast cancerCarcinogenesis: Integrative Cancer Research, 1997
- Identification and prevalence study of 17 allelic variants of the human NAT2 gene in a white populationPharmacogenetics, 1996
- Determination of HumanNAT2Acetylator Genotype by Restriction Fragment-Length Polymorphism and Allele-Specific AmplificationAnalytical Biochemistry, 1995
- A simple salting out procedure for extracting DNA from human nucleated cellsNucleic Acids Research, 1988