DNA methylation and gene silencing in cancer: which is the guilty party?
- 5 August 2002
- journal article
- review article
- Published by Springer Nature in Oncogene
- Vol. 21 (35) , 5380-5387
- https://doi.org/10.1038/sj.onc.1205598
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 62 references indexed in Scilit:
- CpG Islands in vertebrate genomesPublished by Elsevier ,2004
- Hypermethylation trigger of the glutathione-S-transferase gene (GSTP1) in prostate cancer cellsOncogene, 2002
- Aberrant CpG-island methylation has non-random and tumour-type–specific patternsNature Genetics, 2000
- Detailed methylation analysis of the glutathione S-transferase π (GSTP1) gene in prostate cancerOncogene, 1999
- Sp1 sites in the mouse aprt gene promoter are required to prevent methylation of the CpG island.Genes & Development, 1994
- Undermethylation of specific LINE-1 sequences in human cells producing a LINE-1 -encoded proteinGene, 1993
- Targeted mutation of the DNA methyltransferase gene results in embryonic lethalityPublished by Elsevier ,1992
- CpG methylation of the cAMP-responsive enhancer/promoter sequence TGACGTCA abolishes specific factor binding as well as transcriptional activation.Genes & Development, 1989
- Cloning and sequencing of a cDNA encoding DNA methyltransferase of mouse cellsJournal of Molecular Biology, 1988
- X inactivation, differentiation, and DNA methylationCytogenetic and Genome Research, 1975