Mitochondrial fission is an upstream and required event for bax foci formation in response to nitric oxide in cortical neurons
Open Access
- 20 October 2006
- journal article
- Published by Springer Nature in Cell Death & Differentiation
- Vol. 14 (3) , 462-471
- https://doi.org/10.1038/sj.cdd.4402046
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Nitric oxide and neurological disordersMolecular Aspects of Medicine, 2005
- Crosstalk between Nitric Oxide and Zinc Pathways to Neuronal Cell Death Involving Mitochondrial Dysfunction and p38-Activated K+ ChannelsNeuron, 2004
- Mitochondrial Biogenesis in Mammals: The Role of Endogenous Nitric OxideScience, 2003
- p38 Map Kinase Mediates Bax Translocation in Nitric Oxide–Induced Apoptosis in NeuronsThe Journal of cell biology, 2000
- The dynamin-related GTPase Dnm1 regulates mitochondrial fission in yeastNature Cell Biology, 1999
- Nitric Oxide Acutely Inhibits Neuronal Energy ProductionJournal of Neuroscience, 1999
- Chapter 15 Nitric oxide in neurodegenerationPublished by Elsevier ,1998
- Apoptosis and necrosis: two distinct events induced, respectively, by mild and intense insults with N-methyl-D-aspartate or nitric oxide/superoxide in cortical cell cultures.Proceedings of the National Academy of Sciences, 1995
- Nanomolar concentrations of nitric oxide reversibly inhibit synaptosomal respiration by competing with oxygen at cytochrome oxidaseFEBS Letters, 1994
- Inhibition of Mitochondrial Electron Transport by PeroxynitriteArchives of Biochemistry and Biophysics, 1994