Iron-Export Ferroxidase Activity of β-Amyloid Precursor Protein Is Inhibited by Zinc in Alzheimer's Disease
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Open Access
- 2 September 2010
- journal article
- research article
- Published by Elsevier
- Vol. 142 (6) , 857-867
- https://doi.org/10.1016/j.cell.2010.08.014
Abstract
No abstract availableKeywords
This publication has 75 references indexed in Scilit:
- Correlation of iron in the hippocampus with MMSE in patients with Alzheimer's diseaseJournal of Magnetic Resonance Imaging, 2009
- Redox active iron accumulation in aceruloplasminemiaNeuropathology, 2008
- RETRACTED: The Hepcidin-Binding Site on Ferroportin Is Evolutionarily ConservedCell Metabolism, 2008
- Ferroxidase activity is required for the stability of cell surface ferroportin in cells expressing GPI-ceruloplasminThe EMBO Journal, 2007
- High-resolution X-ray Structures of Human Apoferritin H-chain Mutants Correlated with Their Activity and Metal-binding SitesJournal of Molecular Biology, 2007
- Age-Related Changes in Iron Homeostasis and Cell Death in the Cerebellum of Ceruloplasmin-Deficient MiceJournal of Neuroscience, 2006
- Age-related evolution of amyloid burden, iron load, and MR relaxation times in a transgenic mouse model of Alzheimer's diseaseNeurobiology of Disease, 2006
- Iron, brain ageing and neurodegenerative disordersNature Reviews Neuroscience, 2004
- Increased Activity‐Regulating and Neuroprotective Efficacy of α‐Secretase‐Derived Secreted Amyloid Precursor Protein Conferred by a C‐Terminal Heparin‐Binding DomainJournal of Neurochemistry, 1996
- β-amyloid precursor protein-deficient mice show reactive gliosis and decreased locomotor activityCell, 1995