Imidazoline2 (I2) binding site- and α2-adrenoceptor-mediated modulation of central noradrenergic and HPA axis function in control rats and chronically stressed rats with adjuvant-induced arthritis

This publication has 29 references indexed in Scilit:

• Pharmacologic Characterization of Imidazoline Receptor Proteins Identified by Immunologic Techniques and Other Methods^a
  Annals of the New York Academy of Sciences, 1999
• Effect of isolation-rearing on noradrenaline release in rat hypothalamus and hippocampus in vitro
  Brain Research, 1997
• Social isolation in the rat enhances α2-autoreceptor function in the hippocampus in vivo
  Neuroscience, 1997
• Evidence for Altered Control of Hypothalamic CRF in Immune‐Mediated Diseasesa
  Annals of the New York Academy of Sciences, 1995
• Neurotransmitter Regulation of the Hypothalamic Corticotropin‐Releasing Hormone Neuron
  Annals of the New York Academy of Sciences, 1995
• Psychoendocrinologic mechanisms of life stress
  Stress Medicine, 1994
• ROLE OF CENTRAL CATECHOLAMINES IN THE MODULATION OF CORTICOTROPHIN-RELEASING FACTOR mRNA DURING ADJUVANT-INDUCED ARTHRITIS IN THE RAT
  Rheumatology, 1994
• Restraint-stress-induced changes in exploratory behavior appear to be mediated by norepinephrine-stimulated release of CRF
  Journal of Neuroscience, 1989
• Anatomical specificity of noradrenergic inputs to the paraventricular and supraoptic nuclei of the rat hypothalamus
  Journal of Comparative Neurology, 1988
• Corticotropin releasing activity of the new CRF is potentiated several times by vasopressin
  Nature, 1982