Role of G Protein–Coupled Receptor Kinase 2 and Arrestins in β-Adrenergic Receptor Internalization
- 1 July 1998
- journal article
- Published by Elsevier in Trends in Cardiovascular Medicine
- Vol. 8 (5) , 234-240
- https://doi.org/10.1016/s1050-1738(98)00008-5
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Molecular mechanism of desensitization of the chemokine receptor CCR-5: receptor signaling and internalization are dissociable from its role as an HIV-1 co-receptorThe EMBO Journal, 1997
- Mechanism of β-Adrenergic Receptor Desensitization in Cardiac Hypertrophy Is Increased β-Adrenergic Receptor KinaseJournal of Biological Chemistry, 1997
- The Cardiac β-Adrenoceptor–Mediated Signaling Pathway and Its Alterations in Hypertensive Heart DiseaseHypertension, 1997
- Regulatory mechanisms that modulate signalling by G-protein-coupled receptorsBiochemical Journal, 1997
- G protein-coupled receptors: heterologous regulation of homologous desensitization and its implicationsTrends in Pharmacological Sciences, 1996
- The Management of Chronic Heart FailureNew England Journal of Medicine, 1996
- Changes in myocardial and vascular receptors in heart failureJournal of the American College of Cardiology, 1993
- Adrenergic receptors and their signal transduction mechanisms in hypertensionJournal Of Hypertension, 1992
- β-Adrenergic Receptor Kinase: Primary Structure Delineates a Multigene FamilyScience, 1989
- Removal of phosphorylation sites from the β2-adrenergic receptor delays onset of agonist-promoted desensitizationNature, 1988