Developmental evolution of the delayed rectifier current IKs in canine heart appears dependent on the β subunit minK
- 31 December 2004
- journal article
- Published by Elsevier in Heart Rhythm
- Vol. 1 (6) , 704-711
- https://doi.org/10.1016/j.hrthm.2004.08.012
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Developmental changes in and contribute to age-related expression of dofetilide effects on repolarization and proarrhythmiaCardiovascular Research, 2003
- Molecular basis of functional voltage‐gated K+ channel diversity in the mammalian myocardiumThe Journal of Physiology, 2000
- Developmental differences in delayed rectifying outward current in feline ventricular myocytes.American Journal of Physiology-Heart and Circulatory Physiology, 2000
- Roles of the voltage-gated K + channel subunits, Kv 1.5 and Kv 1.4, in the developmental changes of K + currents in cultured neonatal rat ventricular cellsPflügers Archiv - European Journal of Physiology, 1997
- Developmental Changes in the Delayed Rectifier K + Channels in Mouse HeartCirculation Research, 1996
- Developmental cardiac electrophysiology recent advances in cellular physiologyCardiovascular Research, 1996
- A mechanistic link between an inherited and an acquird cardiac arrthytmia: HERG encodes the IKr potassium channelCell, 1995
- Induction of rhythm abnormalities in the fetal rat heart. A tentative mechanism for the embryotoxic effect of the class III antiarrhythmic agent almokalantCardiovascular Research, 1994
- Age-related appearance of outward currents may contribute to developmental differences in ventricular repolarization.Circulation Research, 1992
- Developmental changes in membrane Ca2+ and K+ currents in fetal, neonatal, and adult rabbit ventricular myocytes.Circulation Research, 1992