A model for tumor cell killing by topoisomerase poisons.

  • 1 January 1990
    • journal article
    • review article
    • Vol. 2  (1) , 23-7
Abstract
Our model proposes that topoisomerase-targeting antitumor drugs form reversible drug-enzyme-DNA complexes that collide with the DNA and RNA synthesis machineries. On collision, the complexes lose their reversibility, and generate lethal double-strand DNA breaks. Further investigations of topoisomerase action will allow this model to be refined, and may ultimately lead to the development of more effective anticancer drugs.

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