Insulin resistance syndrome: Defective GABA neuromodulation as a possible hereditary pathogenetic factor (the ‘GABA hypothesis’)
- 31 March 1993
- journal article
- Published by Elsevier in Medical Hypotheses
- Vol. 40 (4) , 197-206
- https://doi.org/10.1016/0306-9877(93)90041-n
Abstract
No abstract availableKeywords
This publication has 54 references indexed in Scilit:
- The GABAA receptor β3 subunit gene: Characterization of a human cDNA from chromosome 15q11q13 and mapping to a region of conserved synteny on mouse chromosome 7Genomics, 1991
- Insulin Resistance: A Multifaceted Syndrome Responsible for NIDDM, Obesity, Hypertension, Dyslipidemia, and Atherosclerotic Cardiovascular DiseaseDiabetes Care, 1991
- 1989 McCollum Award lecture. Genetic and hypothalamic mechanisms for obesity--finding the needle in the haystackThe American Journal of Clinical Nutrition, 1989
- Benzodiazepines and the mammalian retina. I. Autoradiographic localisation of receptor sites and the lack of effect on the electroretinogramBrain Research, 1989
- The rabbit electroretinogram: Effect of GABA and its antagonistsVision Research, 1988
- Wolfram's Syndrome: A Clinical, Diagnostic, and Interpretative ContributionDiabetes Care, 1986
- Chapter 1 GABAergic mechanisms in the retinaProgress in Retinal Research, 1986
- Diabetes mellitus, thiamine-dependentmegaloblastic anemia, and sensorineural deafness associated with deficient α-ketoglutarate dehydrogenase activityThe Journal of Pediatrics, 1985
- Huntington's diseaseNeurology, 1984
- Abnormal activation of private dehydrogenase in Leigh disease fibroblastsNeurology, 1982