Thrombospondin peptides are potent inhibitors of mesangial and glomerular endothelial cell proliferation in vitro and in vivo

This publication has 25 references indexed in Scilit:

• Thrombospondin-1 is required for normal murine pulmonary homeostasis and its absence causes pneumonia.
  Journal of Clinical Investigation, 1998
• Antiproliferative and antitumor activities of D‐reverse peptides derived from the second type‐1 repeat of thrombospondin‐1
  Chemical Biology & Drug Design, 1997
• Thrombospondin 1 is expressed by proliferating mesangial cells and is up-regulated by PDGF and bFGF in vivo
  Kidney International, 1995
• Diversity of function is inherent in matricellular proteins: an appraisal of thrombospondin 1.
  The Journal of cell biology, 1995
• Regulation of Transforming Growth Factor-β Activation by Discrete Sequences of Thrombospondin 1
  Journal of Biological Chemistry, 1995
• The glomerular response to injury: Progression or resolution?
  Kidney International, 1994
• Modulation of endothelial cell proliferation, adhesion, and motility by recombinant heparin‐binding domain and synthetic peptides from the type I repeats of thrombospondin
  Journal of Cellular Biochemistry, 1993
• Thrombospondin causes activation of latent transforming growth factor- beta secreted by endothelial cells by a novel mechanism [published erratum appears in J Cell Biol 1993 Sep;122(5):following 1143]
  The Journal of cell biology, 1993
• Inhibition of fibronectin binding and fibronectin-mediated cell adhesion to collagen by a peptide from the second type I repeat of thrombospondin.
  The Journal of cell biology, 1993
• Complement membrane attack complex stimulates production of reactive oxygen metabolites by cultured rat mesangial cells.
  Journal of Clinical Investigation, 1986