Deregulated GSK3β activity in colorectal cancer: Its association with tumor cell survival and proliferation
- 1 September 2005
- journal article
- clinical trial
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 334 (4) , 1365-1373
- https://doi.org/10.1016/j.bbrc.2005.07.041
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Health, Labour and Welfare
- Japan Society for the Promotion of Science
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 23 references indexed in Scilit:
- Inhibition of glycogen synthase kinase-3 represses androgen receptor activity and prostate cancer cell growthOncogene, 2004
- Pharmacological inhibitors of glycogen synthase kinase 3Trends in Pharmacological Sciences, 2004
- Associations Among -TrCP, an E3 Ubiquitin Ligase Receptor, -Catenin, and NF- B in Colorectal CancerJNCI Journal of the National Cancer Institute, 2004
- Suppression of Androgen Receptor-mediated Transactivation and Cell Growth by the Glycogen Synthase Kinase 3β in Prostate CellsJournal of Biological Chemistry, 2004
- The glamour and gloom of glycogen synthase kinase-3Trends in Biochemical Sciences, 2004
- Identification of a five-pass transmembrane protein family localizing in the Golgi apparatus and the ERBiochemical and Biophysical Research Communications, 2003
- Phosphatidylinositol 3-Kinase/Akt Stimulates Androgen Pathway through GSK3β Inhibition and Nuclear β-Catenin AccumulationJournal of Biological Chemistry, 2002
- Extensive characterization of genetic alterations in a series of human colorectal cancer cell linesOncogene, 2001
- The oncogenic activation of β-cateninCurrent Opinion in Genetics & Development, 1999
- Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase BNature, 1995