Role of Rho/ROCK and p38 MAP Kinase Pathways in Transforming Growth Factor-β-mediated Smad-dependent Growth Inhibition of Human Breast Carcinoma Cells in Vivo
Open Access
- 1 January 2005
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 280 (2) , 1024-1036
- https://doi.org/10.1074/jbc.m403960200
Abstract
No abstract availableKeywords
This publication has 55 references indexed in Scilit:
- Transforming Growth Factor β-Mediated Transcriptional Repression of c-myc Is Dependent on Direct Binding of Smad3 to a Novel Repressive Smad Binding ElementMolecular and Cellular Biology, 2004
- p38 MAP kinase modulates Smad-dependent changes in human prostate cell adhesionOncogene, 2003
- Attenuation of the TGF-β-Smad signaling pathway in pancreatic tumor cells confers resistance to TGF-β-induced growth arrestOncogene, 2003
- Cross‐talk between ERK MAP kinase and Smad‐signaling pathways enhances TGF‐β dependent responses in human mesangial cellsThe FASEB Journal, 2003
- Transcriptional activation of collagenase-3 by transforming growth factor-β1 is via MAPK and Smad pathways in human breast cancer cellsFEBS Letters, 2002
- Smad3 Mediates Transforming Growth Factor-β-induced Collagenase-3 (Matrix Metalloproteinase-13) Expression in Human Gingival FibroblastsJournal of Biological Chemistry, 2002
- The Stress-activated Protein Kinases p38α and JNK1 Stabilize p21Cip1 by PhosphorylationJournal of Biological Chemistry, 2002
- TGF-β signaling: positive and negative effects on tumorigenesisCurrent Opinion in Genetics & Development, 2002
- The Hallmarks of CancerCell, 2000
- TGFβ signaling is necessary for carcinoma cell invasiveness and metastasisCurrent Biology, 1998