Inhibition by mepacrine and p-bromophenacylbromide of phosphoinositide hydrolysis, glucose oxidation, calcium uptake and insulin release in rat pancreatic islets
- 1 August 1984
- journal article
- research article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 33 (16) , 2657-2662
- https://doi.org/10.1016/0006-2952(84)90641-5
Abstract
No abstract availableThis publication has 25 references indexed in Scilit:
- Stimulation of phosphoinositide breakdown in rat pancreatic islets by glucose and carbamylcholineBiochemical and Biophysical Research Communications, 1983
- Regulation of arachidonic acid production: role of phospholipases C and A2Trends in Pharmacological Sciences, 1982
- The enzymology of stimulated inositol lipid turnoverCell Calcium, 1982
- The effects of mepacrine and p-bromophenacyl bromide on arachidonic acid release in human plateletsArchives of Biochemistry and Biophysics, 1982
- Methylation of histidine-48 in pancreatic phospholipase A2. Role of histidine and calcium ion in the catalytic mechanismBiochemistry, 1980
- Mechanisms of inhibition of phospholipase A2Biochemical Pharmacology, 1980
- Anti-inflammatory and platelet anti-aggregant activity of phospholipase-A2 inhibitorsJournal of Pharmacy and Pharmacology, 1979
- The hydrolysis of phosphatidylinositol by lysosomal enzymes of rat liver and brainBiochemical Journal, 1978
- The importance of phospholipase-A2 in prostaglandin biosynthesisBiochemical Pharmacology, 1976
- Method for the Isolation of Intact Islets of Langerhans from the Rat PancreasDiabetes, 1967