All-Trans-Retinoic Acid Induces Interleukin-8 via the Nuclear Factor-κB and p38 Mitogen-Activated Protein Kinase Pathways in Normal Human Keratinocytes
- 1 December 2004
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 123 (6) , 1078-1085
- https://doi.org/10.1111/j.0022-202x.2004.23503.x
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Keratinocyte G2/M Growth Arrest by 1,25-Dihydroxyvitamin D3 Is Caused by Cdc2 Phosphorylation Through Wee1 and Myt1 RegulationJournal of Investigative Dermatology, 2004
- Keratinocyte Growth Inhibition by High-Dose Epidermal Growth Factor Is Mediated by Transforming Growth Factor β Autoinduction: A Negative Feedback Mechanism for Keratinocyte GrowthJournal of Investigative Dermatology, 2003
- Suppressor of Cytokine Signaling 1/JAB and Suppressor of Cytokine Signaling 3/Cytokine-Inducible SH2 Containing Protein 3 Negatively Regulate the Signal Transducers and Activators of Transcription Signaling Pathway in Normal Human Epidermal KeratinocytesJournal of Investigative Dermatology, 2003
- Interleukin‐8, a chemotactic and inflammatory cytokinePublished by Wiley ,2002
- Inhibition of the transcription factor nuclear factor-κB by adenoviral-mediated expression of IκBαM results in tumor cell deathSurgery, 1999
- Current Use and Future Potential Role of Retinoids in DermatologyDrugs, 1997
- Regulation of Interleukin-8 Gene Expression by All-trans Retinoic AcidBiochemical and Biophysical Research Communications, 1995
- Function and Activation of NF-kappaB in the Immune SystemAnnual Review of Immunology, 1994
- Identification of a second human retinoic acid receptorNature, 1988
- A human retinoic acid receptor which belongs to the family of nuclear receptorsNature, 1987