O2 sensing by recombinant TWIK-related halothane-inhibitable K+ channel-1 background K+ channels heterologously expressed in human embryonic kidney cells
- 1 January 2005
- journal article
- Published by Elsevier in Neuroscience
- Vol. 135 (4) , 1087-1094
- https://doi.org/10.1016/j.neuroscience.2005.07.009
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Biophysical characterization of whole-cell currents in O2-sensitive neurons from the rat glossopharyngeal nerveNeuroscience, 2005
- System-specific O2 sensitivity of the tandem pore domain K+ channel TASK-1American Journal of Physiology-Cell Physiology, 2004
- Regulation of recombinant human brain tandem P domain K+channels by hypoxia: a role for O2in the control of neuronal excitability?Journal of Cellular and Molecular Medicine, 2004
- A novel O2-sensing mechanism in rat glossopharyngeal neurones mediated by a halothane-inhibitable background K+ conductanceThe Journal of Physiology, 2003
- Recombinant hTASK1 Is an O2-Sensitive K+ ChannelBiochemical and Biophysical Research Communications, 2001
- Potassium leak channels and the KCNK family of two-p-domain subunitsNature Reviews Neuroscience, 2001
- An oxygen‐, acid‐ and anaesthetic‐sensitive TASK‐like background potassium channel in rat arterial chemoreceptor cellsThe Journal of Physiology, 2000
- O2‐sensitive K+ channels: role of the Kv1.2 α‐subunit in mediating the hypoxic responseThe Journal of Physiology, 2000
- Nitric oxide synthase in the rat carotid body and carotid sinusCell and tissue research, 1994
- Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patchesPflügers Archiv - European Journal of Physiology, 1981