Neuronal toxicity of expanded polyglutamine depends on intracellular distribution in addition to the expression level
- 13 August 2008
- journal article
- Published by Wiley in Neuropathology
- Vol. 28 (5) , 485-496
- https://doi.org/10.1111/j.1440-1789.2008.00908.x
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Pathogenesis of Inclusion Bodies in (CAG)n/Qn-Expansion Diseases with Special Reference to the Role of Tissue Transglutaminase and to Selective VulnerabilityJournal of Neurochemistry, 2008
- Protein Quality Control in Alzheimers Disease: A Fatal SaviourCurrent Drug Targets-CNS & Neurological Disorders, 2005
- Protein Aggregation in Alzheimers Disease and Other Neoropathological DisordersCurrent Alzheimer Research, 2005
- Beyond the Qs in the polyglutamine diseases: Table 1.Genes & Development, 2001
- Oxidation versus aggregation — how do SOD1 mutants cause ALS?Nature Medicine, 2000
- Tau protein isoforms, phosphorylation and role in neurodegenerative disorders11These authors contributed equally to this work.Brain Research Reviews, 2000
- Bunina bodies in amyotrophic lateral sclerosis on Guam: a histochemical, immunohistochemical and ultrastructural investigation.Acta Neuropathologica, 1999
- Pathogenic implications of mutations in the tau gene in pallido-ponto-nigral degeneration and related neurodegenerative disorders linked to chromosome 17Proceedings of the National Academy of Sciences, 1998
- Genetics of Parkinson's diseaseHuman Molecular Genetics, 1997
- Mutation in the α-Synuclein Gene Identified in Families with Parkinson's DiseaseScience, 1997