Cyclic stretch induces the release of growth promoting factors from cultured neonatal cardiomyocytes and cardiac fibroblasts
- 1 January 2000
- journal article
- Published by Springer Nature in Molecular and Cellular Biochemistry
- Vol. 208 (1/2) , 89-98
- https://doi.org/10.1023/a:1007046105745
Abstract
Growth factors and hormones may play an autocrine/paracrine role in mechanical stress-induced cardiac hypertrophy. Using an in vitromodel of mechanical stress, i.e. stretch of cardiomyocytes and...Keywords
This publication has 36 references indexed in Scilit:
- Cultured neonatal rat cardiac myocytes and fibroblasts do not synthesize renin or angiotensinogen evidence for stretch-induced cardiomyocyte hypertrophy independent of angiotensin IICardiovascular Research, 1999
- Angiotensin II stimulates cardiac myocyte hypertrophy via paracrine release of TGF-β1 and endothelin-1 from fibroblastsCardiovascular Research, 1998
- Stretch-induced VEGF expression in the heart.Journal of Clinical Investigation, 1997
- Intermediate Filament-mediated Stretch-induced Changes in Chromatin: A Hypothesis for Growth Initiation in Cardiac MyocytesJournal of Molecular and Cellular Cardiology, 1996
- Endothelin-1 Is Involved in Mechanical Stress-induced Cardiomyocyte HypertrophyJournal of Biological Chemistry, 1996
- Hypertrophic stimuli induce transforming growth factor-beta 1 expression in rat ventricular myocytes.Journal of Clinical Investigation, 1994
- Autocrine release of angiotensin II mediates stretch-induced hypertrophy of cardiac myocytes in vitroCell, 1993
- Endothelin-1 is an autocrine/paracrine factor in the mechanism of angiotensin II-induced hypertrophy in cultured rat cardiomyocytes.Journal of Clinical Investigation, 1993
- Signal transduction from the extracellular matrix.The Journal of cell biology, 1993
- Hemodynamic versus adrenergic control of cat right ventricular hypertrophy.Journal of Clinical Investigation, 1985