Mutational analysis of Drosophila synaptotagmin demonstrates its essential role in Ca2+-activated neurotransmitter release
Open Access
- 24 September 1993
- journal article
- research article
- Published by Elsevier
- Vol. 74 (6) , 1125-1134
- https://doi.org/10.1016/0092-8674(93)90733-7
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Tetanus toxin action: Inhibition of neurotransmitter release linked to synaptobrevin proteolysisPublished by Elsevier ,2004
- Synaptotagmin: A calcium-sensitive inhibitor of exocytosis?Cell, 1993
- Synaptic transmission persists in synaptotagmin mutants of DrosophilaCell, 1993
- Synaptic vesicle proteins in exocytosis: what do we know?Current Opinion in Neurobiology, 1993
- SNAP receptors implicated in vesicle targeting and fusionNature, 1993
- Tetanus and botulinum-B neurotoxins block neurotransmitter release by proteolytic cleavage of synaptobrevinNature, 1992
- Action potentials must admit calcium to evoke transmitter releaseNature, 1991
- Altered Synaptic Transmission inDrosophila HyperkineticMutantsJournal of Neurogenetics, 1989
- Calcium ions, active zones and synaptic transmitter releaseTrends in Neurosciences, 1988
- Synaptic vesicle exocytosis captured by quick freezing and correlated with quantal transmitter release.The Journal of cell biology, 1979