β-bungarotoxin-induced depletion of synaptic vesicles at the mammalian neuromuscular junction
- 11 June 2004
- journal article
- Published by Elsevier in Neuropharmacology
- Vol. 47 (2) , 304-314
- https://doi.org/10.1016/j.neuropharm.2004.04.012
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- β‐Bungarotoxin is a potent inducer of apoptosis in cultured rat neurons by receptor‐mediated internalizationEuropean Journal of Neuroscience, 2001
- Monitoring of Black Widow Spider Venom (BWSV) induced exo- and endocytosis in living frog motor nerve terminals with FM1-43Neuropharmacology, 1995
- Exocytotic Ca2+ channels in mammalian central neuronsTrends in Neurosciences, 1995
- Roles of N-Type and Q-Type Ca 2+ Channels in Supporting Hippocampal Synaptic TransmissionScience, 1994
- A new conus peptide ligand for mammalian presynaptic Ca2+ channelsNeuron, 1992
- .beta.-Bungarotoxin-mediated liposome fusion: spectroscopic characterization by fluorescence and ESRBiochemistry, 1990
- Selectivity and Regulation in the Phospholipase A2‐Mediated Attack on Cholinergic Synaptic Vesicles by β‐BungarotoxinJournal of Neurochemistry, 1986
- Effects of black widow spider venom and Ca2+ on quantal secretion at the frog neuromuscular junction.The Journal of general physiology, 1986
- Freeze-fracture studies of frog neuromuscular junctions during intense release of neurotransmitter. I. Effects of black widow spider venom and Ca2+-free solutions on the structure of the active zone.The Journal of cell biology, 1979
- Acute muscle denervation induced by β-bungarotoxinProceedings of the Royal Society of London. B. Biological Sciences, 1976