Through Induction of Juxtaposition and Tyrosine Kinase Activity of Jak1, X-Gene Product of Hepatitis B Virus Stimulates Ras and the Transcriptional Activation through AP-1, NF-κB, and SRE Enhancers
- 7 September 2001
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 286 (5) , 886-894
- https://doi.org/10.1006/bbrc.2001.5496
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- STATs in oncogenesisOncogene, 2000
- Interferonγ Activation of Raf-1 Is Jak1-dependent and p21ras-independentJournal of Biological Chemistry, 1998
- Membrane recruitment of Raf-1 is not the only function of Ras in Raf-1 activationOncogene, 1997
- STATs and Gene RegulationScience, 1997
- Involvement of Janus Kinases in the Insulin Signaling PathwayEuropean Journal of Biochemistry, 1995
- JAK2, Ras, and Raf Are Required for Activation of Extracellular Signal-regulated Kinase/Mitogen-activated Protein Kinase by Growth HormonePublished by Elsevier ,1995
- X-gene Product Antagonizes the p53-mediated Inhibition of Hepatitis B Virus Replication through Regulation of the Pregenomic/Core PromoterJournal of Biological Chemistry, 1995
- Phosphorylation and Activation of the DNA Binding Activity of Purified Stat1 by the Janus Protein-tyrosine Kinases and the Epidermal Growth Factor ReceptorJournal of Biological Chemistry, 1995
- Erythropoietin-induced Recruitment of Shc via a Receptor Phosphotyrosine-independent, Jak2-associated PathwayPublished by Elsevier ,1995
- HBx gene of hepatitis B virus induces liver cancer in transgenic miceNature, 1991