Association of low-density lipoprotein with particulate connective tissue matrix components enhances cholesterol accumulation in cultured subendothelial cells of human aorta
- 1 May 1987
- journal article
- research article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
- Vol. 928 (3) , 251-258
- https://doi.org/10.1016/0167-4889(87)90183-2
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Complexes of low-density lipoproteins and arterial proteoglycan aggregates promote cholesteryl ester accumulation in mouse macrophagesBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1985
- Lipids in cells of atherosclerotic and uninvolved human aortaExperimental and Molecular Pathology, 1985
- Lipoprotein-heparin-fibronectin-denatured collagen complexes enhance cholesteryl ester accumulation in macrophages.The Journal of cell biology, 1984
- Lipid accumulation in arterial smooth muscle cells in culture Morphological and biochemical changes caused by low density lipoproteins and chloroquineAtherosclerosis, 1982
- Cellular and Molecular Biology of Lipoprotein Metabolism in AtherosclerosisDiabetes, 1981
- Flux of plasma lipoproteins into human arterial intima Comparison between grossly normal and atheromatous intimaAtherosclerosis, 1981
- Reversible accumulation of cholesteryl esters in macrophages incubated with acetylated lipoproteins.The Journal of cell biology, 1979
- Altered Metabolism (In Vivo and In Vitro) of Plasma Lipoproteins after Selective Chemical Modification of Lysine Residues of the ApoproteinsJournal of Clinical Investigation, 1979
- The Relationship Between Plasma and Tissue Lipids in Human AtherosclerosisPublished by Elsevier ,1974
- Arterial Composition and Metabolism: Esterified Fatty Acids and Cholesterol1 1Publication No. 413 from the Oregon Regional Primate Research Center, supported in part by Grant No. FR 00163 of the National Institutes of Health. The experiments carried out in our laboratory were supported by a grant-in-aid from the National Institutes of Health (HE 09744), Bethesda, Maryland.Published by Elsevier ,1970