Synergy or Independence? Deciphering the Interaction of HLA Class I and NK Cell KIR Alleles in Early HIV-1 Disease Progression
Open Access
- 20 April 2007
- journal article
- review article
- Published by Public Library of Science (PLoS) in PLoS Pathogens
- Vol. 3 (4) , e43
- https://doi.org/10.1371/journal.ppat.0030043
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- KIR/HLA Pleiotropism: Protection against Both HIV and Opportunistic InfectionsPLoS Pathogens, 2006
- Killer immunoglobulin-like receptors and HLA act both independently and synergistically to modify HIV disease progressionGenes & Immunity, 2005
- HANGING IN THE BALANCE: KIR and Their Role in DiseaseMolecular Interventions, 2005
- Distinct Mechanisms of CD4+and CD8+T-Cell Activation and Bystander Apoptosis Induced by Human Immunodeficiency Virus Type 1 VirionsJournal of Virology, 2005
- Immune activation set point during early HIV infection predicts subsequent CD4+ T-cell changes independent of viral loadBlood, 2004
- The Influence ofHLAGenotype on AIDSAnnual Review of Medicine, 2003
- Epistatic interaction between KIR3DS1 and HLA-B delays the progression to AIDSNature Genetics, 2002
- Effect of a Single Amino Acid Change in MHC Class I Molecules on the Rate of Progression to AIDSNew England Journal of Medicine, 2001
- HLA-derived peptides which inhibit T cell function bind to members of the heat-shock protein 70 family.The Journal of Experimental Medicine, 1996
- NK3-specific natural killer cells are selectively inhibited by Bw4-positive HLA alleles with isoleucine 80.The Journal of Experimental Medicine, 1994