Differential Effects of Nonselective Nitric Oxide Synthase (NOS) and Selective Inducible NOS Inhibition on Hepatic Necrosis, Apoptosis, ICAM-1 Expression, and Neutrophil Accumulation during Endotoxemia
- 31 October 1997
- journal article
- Published by Elsevier in Nitric Oxide
- Vol. 1 (5) , 404-416
- https://doi.org/10.1006/niox.1997.0136
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Release of soluble intercellular adhesion molecule 1 into bile and serum in murine endotoxin shockHepatology, 1996
- Interleukin-1 and nitric oxide protect against tumor necrosis factor α-induced liver injury through distinct pathwaysHepatology, 1995
- Nitrogen oxide‐induced autoprotection in isolated rat hepatocytesFEBS Letters, 1995
- INHIBITION OF NITRIC OXIDE SYNTHESIS AGGRAVATES REPERFUSION INJURY AFTER HEPATIC ISCHEMIA AND ENDOTOXEMIAShock, 1995
- Endotoxin-stimulated nitric oxide production increases injury and reduces rat liver chemiluminescence during reperfusionGastroenterology, 1995
- Nitric oxide produced by human B lymphocytes inhibits apoptosis and Epstein-Barr virus reactivationCell, 1994
- Nitric Oxide Synthase Expression Is Induced in Hepatocytes in Vivo during Hepatic InflammationJournal of Surgical Research, 1993
- Hepatocyte Injury by Activated Neutrophils In Vitro Is Mediated by ProteasesAnnals of Surgery, 1993
- Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures.Journal of Histochemistry & Cytochemistry, 1981
- Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activationNature, 1980