The lack of effect of specific overexpression of IGF-1 in the central nervous system or skeletal muscle on pathophysiology in the G93A SOD-1 mouse model of ALS
- 2 June 2007
- journal article
- Published by Elsevier in Experimental Neurology
- Vol. 207 (1) , 52-63
- https://doi.org/10.1016/j.expneurol.2007.05.016
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Neuromuscular Development in the Absence of Programmed Cell Death: Phenotypic Alteration of Motoneurons and MuscleJournal of Neuroscience, 2006
- Transgenic mouse models of amyotrophic lateral sclerosisBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2006
- IGF-I specifically enhances axon outgrowth of corticospinal motor neuronsNature Neuroscience, 2006
- Increased expression of insulin-like growth factor-I (IGF-I) during embryonic development produces neocortical overgrowth with differentially greater effects on specific cytoarchitectonic areas and cortical layersDevelopmental Brain Research, 2005
- Target-Derived Cardiotrophin-1 and Insulin-like Growth Factor-I Promote Neurite Growth and Survival of Developing Oculomotor NeuronsMolecular and Cellular Neuroscience, 2002
- Selective Loss of α Motoneurons Innervating the Medial Gastrocnemius Muscle in a Mouse Model of Amyotrophic Lateral SclerosisExperimental Neurology, 1998
- In vivo actions of insulin-like growth factor-I (IGF-I) on cerebellum development in transgenic mice: evidence that IGF-I increases proliferation of granule cell progenitorsDevelopmental Brain Research, 1996
- Neurotrophic agents prevent motoneuron death following sciatic nerve section in the neonatal mouseJournal of Neurobiology, 1994
- Activity‐Sensitive Signaling by Muscle‐Derived Insulin‐like Growth Factors in the Developing and Regenerating Neuromuscular SystemAnnals of the New York Academy of Sciences, 1993
- Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosisNature, 1993